vtioВестник трансплантологии и искусственных органовRussian Journal of Transplantology and Artificial Organs1995-1191Academician V.I.Shumakov National Medical Research Center of Transplantology and Artificial Organs", Ministry of Health of the Russian Federation10.15825/1995-1191-2012-2-32-42vtio-161Research ArticleКлиническая трансплантологияClinical TransplantologyОЦЕНКА СОСТОЯНИЯ МАКРОМОЛЕКУЛЯРНОЙ СТРУКТУРЫ КАРДИОМИОЦИТОВ У БОЛЬНЫХ РАЗЛИЧНЫМИ ФОРМАМИ КАРДИОМИОПАТИИEVALUATION OF THE MACROMOLECULAR STRUCTURE OF CARDIAC MYOCYTES IN PATIENTS WITH VARIOUS FORMS OF CARDIOMYOPATHYКуприяноваА. Г.KupriyanovaA. G.БелецкаяЛ. В.BeletskayaL. V.ЗайденовВ. А.ZaidenovV. A.СаитгареевР. Ш.SaitgareevR. Sh.ГольцА. М.GoltsA. M.ЗахаревичВ. М.ZakharevichV. M.ГотьеС. В.GautierS. V.ФГБУ «ФНЦ трансплантологии и искусственных органов им. академика В.И. Шумакова» Минздравсоцразвития России, г. МоскваAcademican V.I. Shumakov Federal Research Center of Transplantology and Artificial Organs, Moscow201228052014142ВЕСТНИК ТРАНСПЛАНТОЛОГИИ И ИСКУССТВЕННЫХ ОРГАНОВ том XIV No 2–20123242Copyright © Куприянова А.Г., Белецкая Л.В., Зайденов В.А., Саитгареев Р.Ш., Гольц А.М., Захаревич В.М., Готье С.В., 20122012Куприянова А.Г., Белецкая Л.В., Зайденов В.А., Саитгареев Р.Ш., Гольц А.М., Захаревич В.М., Готье С.В.Kupriyanova A.G., Beletskaya L.V., Zaidenov V.A., Saitgareev R.S., Golts A.M., Zakharevich V.M., Gautier S.V.Данная работа распространяется под лицензией Creative Commons Attribution 4.0.This work is licensed under a Creative Commons Attribution 4.0 License.https://journal.transpl.ru/vtio/article/view/161

При оценке состояния структурных белков кардиомиоцитов у больных различными формами кардиомио- патии (49 образцов миокарда) выявлены изменения, характерные для дилатационной кардиомиопатии, а также общие нарушения молекулярной структуры, развивающиеся вне зависимости от этиологии за- болевания. 

Assessing the condition of the structural proteins of cardiomyocytes in patients with various forms of cardiomyo- pathy (49 samples) revealed changes characteristic for dilated cardiomyopathy, as well as common disorders of the molecular structure, developing, regardless of the etiology of the disease. 

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The authors declare that there are no conflicts of interest present.